In our research we focus on the genetic causes of atherosclerosis and its effects on plaque morphology that underly ischemic stroke and coronary artery disease, and subclinical atherosclerosis of the carotid and coronaries.

With our collaborative work we aim to move from genome-wide association studies loci to novel therapeutic targets and (surrogate) biomarkers for cardiovascular diseases through extensive in silico, and in vitro modelling.

We support and promote principles of Open Science by letting values of quality and integrity, science for the benefit of all, fairness, equity, diversity and inclusiveness guide our work. We value transparency, reproducibility, and critique, accountability, responsibility, and collaboration. The Future of Science is Open, as is our Science.